They suggest feasible explanations which include an accumulation of potassium in locks cells, or the as well unexpected removal of potassium. Whatever the research findings Unfortunately, there remains no real way of identifying those at risk for hearing loss or for preventing it. The Royal National Institute for Deaf People which funded the task, says the study does provide expect the treating hearing loss and in addition provides another important little bit of the jigsaw in highlighting a gene associated with age-related hearing loss.The mutation could, for instance, link hypertension to the age-related decline in mitochondrial function, that was determined by Shulman. Shulman’s work shows that this decline can lead to insulin resistance, a major contributor to type 2 diabetes. Lifton does not have any illusions that defining the mechanisms underlying such linkages will be easy. He speculated that the genetic defect may make clinical pathologies by crippling the energy-production capacity for the mitochondria. Alternately, it might raise the production of reactive oxygen species that cause wear and tear on blood vessels, contributing to high blood circulation pressure and other problems. Differentiating between the two possibilities is important, he said, because each indicate a very different therapeutic strategy.